Overview

Stroke is an abrupt interruption of constant blood flow to the brain that causes loss of neurological function. The interruption of blood flow can be caused by a blockage, leading to the more common ischemic stroke, or by bleeding in the brain, leading to the more deadly hemorrhagic stroke. Ischemic stroke constitutes an estimated 87 percent of all stroke cases. Stroke often occurs with little or no warning, and the results can be devastating.

It is crucial that proper blood flow and oxygen be restored to the brain as soon as possible. Without oxygen and important nutrients, the affected brain cells are either damaged or die within a few minutes. Once brain cells die, they generally do not regenerate, and devastating damage may occur, sometimes resulting in physical, cognitive, and mental disabilities.

Types


There are two types of stroke.

Ischemic Stroke 

  • Thrombotic (cerebral thrombosis) is the most common type of ischemic stroke. A blood clot forms inside a diseased or damaged artery in the brain resulting from atherosclerosis (cholesterol-containing deposits called plaque), blocking blood flow.
  • Embolic (cerebral embolism) is caused when a clot or a small piece of plaque formed in one of the arteries leading to the brain or in the heart, is pushed through the bloodstream and lodges in narrower brain arteries. The blood supply is cut off from the brain due to the clogged vessel.


Hemorrhagic Stroke

  • Subarachnoid Hemorrhage: bleeding that occurs in the space between the surface of them brain and the skull. A common cause of subarachnoid hemorrhagic stroke is a ruptured cerebral aneurysm, an area where a blood vessel in the brain weakens, resulting in a bulging or ballooning out of part of the vessel wall; or the rupture of an arteriovenous malformation, a tangle of abnormal and poorly formed blood vessels (arteries and veins), with an innate propensity to bleed.
  • Intracerebral Hemorrhage: bleeding that occurs within the brain tissue. Many intracerebral hemorrhages are due to changes in the arteries caused by long-term hypertension. Other potential causes may be delineated through testing.

Specialty care

 

Care team

Profiles

Laura Healy, MD

Vascular Surgeon Surgery | Vascular
747 N. Rutledge Street 4th Floor Springfield, IL 62702
Profiles

Tian Zhang, MD

Vascular Surgeon Surgery | Vascular
747 N. Rutledge Street 4th Floor Springfield, IL 62702
Profiles

William Robinson, MD

Vascular Surgeon Surgery | Vascular
747 N. Rutledge Street 4th Floor Springfield, IL 62702
Profiles

Hesham Allam, MD

Neurologist Neurology | Neurocritical Care
751 N. Rutledge St. Suite 3100 Springfield, IL 62702
Profiles

Janae Ballard, DNP, FNP-C

Nurse Practitioner Neurosurgery
747 N. Rutledge Street 2nd floor Springfield, IL 62702
Profiles

Dale Korinek, Psy.D

Clinical Neuropsychologist Psychiatry
319 E Madison St 3rd Floor Springfield, IL 62702
Profiles

Teah Qvavadze, MD

Vascular Surgeon Surgery | Vascular
747 N. Rutledge Street 4th Floor Springfield, IL 62702
Profiles

Sajjad Mueed, MD

Director, Memorial Medical Center Stroke Center Neurology
751 N. Rutledge St. Suite 3100 Springfield, IL 62702
Profiles

Sydney DeSollar, DNP, FNP-C

Nurse Practitioner Neurosurgery
747 N. Rutledge Street 2nd floor Springfield, IL 62702
Profiles

Nicole Atwood, AGACNP-BC

Advance Practice Nurse Neurology | Neurocritical Care
751 N. Rutledge St. Suite 3100 Springfield, IL 62702
Profiles

Deidra Frisbie, DNP, FNP-BC

Certified Nurse Practitioner Neurology
751 N. Rutledge St. Suite 3100 Springfield, IL 62702

 

Care facilities

Locations
Locations

 

Clinical trials

Trial
Surgery

LeAAPS

Active not recruiting

Left Atrial Appendage Exclusion for Prophylactic Stroke Reduction

Trial
Neuroscience Institute

Sleep for Stroke Management and Recovery Trial

Active recruiting

The primary goals of this study are to determine whether treatment of obstructive sleep apnea with positive airway pressure starting shortly after accute ischemic stroke or high risk TIA (1) reduces recurrent stroke, acute coronary syndrome, and all-cause mortality 6 months after the event and (2) improves stroke outcomes at 3 months in patients who experienced ischemic stroke.